EPIDEMIOLOGICAL CHARACTERISTICS OF PANCREATIC LIPOMATOSIS, HYPERCHOLESTEROLEMIA, AND ALKALINE PHOSPHATASE AS RISK FACTORS FOR BILIARY PANCREATITIS

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Суюнов . D. (2025). EPIDEMIOLOGICAL CHARACTERISTICS OF PANCREATIC LIPOMATOSIS, HYPERCHOLESTEROLEMIA, AND ALKALINE PHOSPHATASE AS RISK FACTORS FOR BILIARY PANCREATITIS. Журнал мультидисциплинарных наук и инноваций, 1(6), 416–419. извлечено от https://www.inlibrary.uz/index.php/jmsi/article/view/135652
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volume 4, issue 7, 2025

416

EPIDEMIOLOGICAL CHARACTERISTICS OF PANCREATIC LIPOMATOSIS,

HYPERCHOLESTEROLEMIA, AND ALKALINE PHOSPHATASE AS RISK

FACTORS FOR BILIARY PANCREATITIS

Suyunov Dilmurod Muminovich

Andijan State Medical Institute

Introduction.

Global studies have established that in more than 40% of cases, pathologies of the biliary tract

play a significant role in the development of pancreatitis. In such situations, pancreatitis arises

against the background of the following conditions: anomalies in the position and shape of the

gallbladder, strictures and cysts of the distal pancreatic duct and common bile duct, stenosis of

the sphincter of Oddi (in 52% of cases), chronic cholecystitis (in 42%), gallstone disease or

consequences of cholecystectomy (in 6%) (Tsuman I.G. et al., 2001). Up to 25% of cases of

pancreatitis in children are caused by gastrointestinal disorders (Moriville V.D. et al., 2010;

Kornienko E.A. et al., 2010).

According to data presented by Rizaev K.S., Shukurov B.I., and Sattarov B.S. (2022), in 20% of

patients with acute pancreatitis, pancreatic necrosis is diagnosed, which increases mortality to

20–30% due to infection of pancreatic necrosis, developing in 70% of cases.

Studies by Capp J. et al. (2019), Wadhwa V. et al. (2017), and Petrov M.S. et al. (2010) report

that mortality in acute pancreatitis is around 1%. However, in complicated and severe cases (with

pancreatic necrosis, multiple organ failure, or other complications), this rate can reach 40% or

increase up to 40-fold.

These data clearly demonstrate that biliary pancreatitis is a serious medical, economic, and social

problem. In patients with biliary tract disorders, multiple factors contribute to the development of

biliary pancreatitis.

Objective.

To study the epidemiological characteristics of pancreatic lipomatosis, hypercholesterolemia,

and elevated serum alkaline phosphatase levels as risk factors for biliary pancreatitis.

Materials and Methods.

This study investigated the epidemiological characteristics of the relationship between

hypercholesterolemia (HCh), pancreatic lipomatosis (PL), and elevated serum alkaline

phosphatase (ALP) levels with the development of biliary pancreatitis in 1500 geriatric patients

diagnosed with biliary pancreatitis.

Hypercholesterolemia was identified in one out of every four (25.0%) of the studied population

aged 60 to 90 years.

The prevalence of biliary pancreatitis among elderly individuals with confirmed

hypercholesterolemia was 41.0%, while among those without hypercholesterolemia it was only


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volume 4, issue 7, 2025

417

5.3%. Thus, the prevalence of biliary pancreatitis in the presence of hypercholesterolemia was

more than eight times higher (

p

= 0.012).

The frequency of biliary pancreatitis across different age groups among individuals with and

without hypercholesterolemia is presented below.

Table 1.

Characteristics of the occurrence of biliary pancreatitis depending on the presence of

hypercholesterolemia

OR = 8,11; CI [5,59 – 11,77]; χ² = 154,43; p < 0,05

Pancreatic lipomatosis is reliably associated with an increased risk of developing biliary

pancreatitis [OR = 7.89].

The confidence interval and Fisher’s

p

-value also indicate the

statistical significance of the obtained results [95% CI: 5.48–11.35;

p

< 0.05].

Table 2. Epidemiological characteristics of the prevalence of pancreatic lipomatosis among

elderly individuals and its association with biliary pancreatitis

1.

Among individuals aged

60–74 years

, the prevalence of biliary pancreatitis was

40.0%

in the presence of hypercholesterolemia (HCh) and

6.1%

in its absence, indicating a

6.6-fold

higher prevalence

(

p

= 0.001).

2.

In the age group

75–89 years

, the rates were

42.9%

versus

3.5%

, i.e., more than

14

times higher

in the presence of HCh (

p

= 0.001).

3.

In individuals older than

90 years

, biliary pancreatitis was also diagnosed significantly

more often in the presence of HCh (

52.2%

) compared with its absence (

20.0%

).

Thus, the prevalence of biliary pancreatitis in the geriatric population with hypercholesterolemia

ranged from

41.0% to 52.2%

, significantly exceeding the rates observed in the control groups

[OR = 8.11; 95% CI: 9.59–11.77; χ² = 154.43;

p

< 0.05].


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The epidemiological characteristics of the prevalence of

pancreatic lipomatosis

in the geriatric

population and its association with the development of biliary pancreatitis were also assessed.

Specific trends and features were confirmed and quantitatively presented in the form of an

analytical review of the obtained data.

Table 3

presents the epidemiological data on the prevalence of elevated alkaline phosphatase

(ALP) levels in elderly individuals and its relationship with biliary pancreatitis. According to the

table, the prevalence of biliary pancreatitis among individuals with elevated ALP (ALP+) was

33.3%

, while in those with normal ALP levels (ALP–) it was

24.1%

, i.e., the difference was

9.2%

(

p

= 0.726), which is not statistically significant.

In the

60–74 years

age group, biliary pancreatitis was observed in

34.4%

(ALP+) and

23.3%

(ALP–),

which

was

statistically

significant

(

p

<

0.05).

In the

≥90 years

age group, the rates were

30.3%

versus

60.0%

respectively (

p

< 0.05), which,

despite the reverse trend, also reached statistical significance.

Table 3. Epidemiological characteristics of the prevalence of alkaline phosphatasemia in

elderly individuals and its association with biliary pancreatitis.

RR = 1,73; CI [1,35 – 2,22]; χ² = 19,36;

p

< 0,05

An elevated level of alkaline phosphatase is reliably associated with an increased risk of

developing biliary pancreatitis up to 73% [OR = 1.73].

The confidence interval and Fisher’s

p

-value also confirm the statistical significance of this result [95% CI: 1.35–2.22;

p

< 0.05].

Conclusions

1.

Hypercholesterolemia

is one of the leading risk factors for the development of biliary

pancreatitis: it was established that in individuals with elevated cholesterol levels, the probability

of developing biliary pancreatitis is

8 times higher

compared to those with normal cholesterol

[OR = 8.11]. The confidence interval and Fisher’s

p

-value also confirm the statistical

significance of this result [95% CI: 5.59–11.77;

p

< 0.05].

2.

Pancreatic lipomatosis (PL)

occurs in

37.0%

of the geriatric population of the Fergana

region. Among individuals without signs of PL, biliary pancreatitis is diagnosed in only

6.4%

of

cases. Thus, the presence of lipomatosis increases the risk of developing biliary pancreatitis by

more than

six-fold

[RR = 7.89; 95% CI: 5.48–11.35; χ² = 155.9;

p

< 0.05].

3.

Elevated alkaline phosphatase (ALP+)

is a reliable risk factor for biliary pancreatitis in

elderly individuals — the disease is diagnosed in

one out of every three patients

with this

biochemical marker [RR = 1.73; 95% CI: 1.35–2.22; χ² = 19.36;

p

< 0.05].


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volume 4, issue 7, 2025

419

Literature:

1. Akinosoglou K., Gogos C. Immune-modulating therapy in acute pancreatitis: fact or fiction.

// World journal of gastroenterology. 2014; 20(41):15200-15212.

2. Bang J.Y., Arnoletti J.P., Holt B.A. Sutton B., Hasan M.K., Navaneethan U., et al. An

endoscopic transluminal approach, compared with minimally invasive surgery, reduces

complications and costs for patients with necrotizing pancreatitis

3. Bellin M.D., Whitcomb D.S., Abberbock J. et al. Patient and disease characteristics

associated wich the presence of diabetes mellitus in adulta with chronic pancreatitis in t the

United states / Am J of Gastroenterol. - 2017; 112(9):1438-1461.

4. Bliss L.A., Yang C.J., Eskander M.F., et al. Surgical management of chronic pancreatitis:

current utilization in the United States. HPB (Oxford). 2015; 17(9):804-810

5. Boxhoorn L., Voermans R.P., Bouwense S.A., Bruno M.J., Verdonk R.C., Boermeester

M.A., van Santvoort H.C., Besselink M.G. Acute pancreatitis. // Lancet. 2020; 396:726-731.

6. Bruce J., Sanchez-Alvarez R., Sans M.D., Sugden S.A., Qi N., James A.D., Williams J.A.

Insulin protects acinar cells during pancreatitis by preserving glycolytic ATP supply to

calcinum pumps. Nature communications. 2021; 12(1):4384.

7. Connor S. Defining post-operative pancreatitis as a new pancreatic specific complication

following pancreatic resection. HPB (oxford). 2016; 18(8) 642:651

8. Freeman M.L., Werner J., van Santvoort H.C., et al. Intervetions for necrotizing pancreatitis:

summary of a multidisciplinary consensus conference. // Pancreas 2012; 41(8):1176-1194

Библиографические ссылки

Akinosoglou K., Gogos C. Immune-modulating therapy in acute pancreatitis: fact or fiction. // World journal of gastroenterology. 2014; 20(41):15200-15212.

Bang J.Y., Arnoletti J.P., Holt B.A. Sutton B., Hasan M.K., Navaneethan U., et al. An endoscopic transluminal approach, compared with minimally invasive surgery, reduces complications and costs for patients with necrotizing pancreatitis

Bellin M.D., Whitcomb D.S., Abberbock J. et al. Patient and disease characteristics associated wich the presence of diabetes mellitus in adulta with chronic pancreatitis in t the United states / Am J of Gastroenterol. - 2017; 112(9):1438-1461.

Bliss L.A., Yang C.J., Eskander M.F., et al. Surgical management of chronic pancreatitis: current utilization in the United States. HPB (Oxford). 2015; 17(9):804-810

Boxhoorn L., Voermans R.P., Bouwense S.A., Bruno M.J., Verdonk R.C., Boermeester M.A., van Santvoort H.C., Besselink M.G. Acute pancreatitis. // Lancet. 2020; 396:726-731.

Bruce J., Sanchez-Alvarez R., Sans M.D., Sugden S.A., Qi N., James A.D., Williams J.A. Insulin protects acinar cells during pancreatitis by preserving glycolytic ATP supply to calcinum pumps. Nature communications. 2021; 12(1):4384.

Connor S. Defining post-operative pancreatitis as a new pancreatic specific complication following pancreatic resection. HPB (oxford). 2016; 18(8) 642:651

Freeman M.L., Werner J., van Santvoort H.C., et al. Intervetions for necrotizing pancreatitis: summary of a multidisciplinary consensus conference. // Pancreas 2012; 41(8):1176-1194

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